| Vav3 oncogene is overexpressed and regulates cell growth and androgen receptor activity in human prostate cancer development and progression. | prostate, |
| Vav3 oncogene is overexpressed and regulates cell growth and androgen receptor activity in human prostate cancer. | prostate, |
| Vav3 oncogene activates estrogen receptor and its overexpression may be involved in human breast cancer. | breast, |
| Targeted overexpression of vav3 oncogene in prostatic epithelium induces nonbacterial prostatitis and prostate cancer. | prostate, |
| The molecular mechanism of Vav3 oncogene on upregulation of androgen receptor activity in prostate cancer cells. | prostate, |
| Vav3 oncogene is involved in regulation of secretory phospholipase A2-IIa expression in prostate cancer. | prostate, |
| Data indicate that Vav3 oncogene protein plays a crucial role in prostate cancer growth and malignant behavior, and could be a potential therapeutic target. | prostate, |
| The Vav3 oncogene enhances the malignant potential of prostate cancer cells under chronic hypoxia. | prostate, |
| Targeting the Vav3 oncogene enhances docetaxel-induced apoptosis through the inhibition of androgen receptor phosphorylation in LNCaP prostate cancer cells under chronic hypoxia. | prostate, |
| Vav3 is involved in the proliferation, migration, and invasion of gastric cancer cell as a tumor oncogene | gastric, |
| VAV3 oncogene expression in colorectal cancer: clinical aspects and functional characterization. | colorectal, |
| Differential Gene Expression of the Proto-oncogene VAV3 and the Transcript Variant VAV3.1 in Oral Squamous Cell Carcinoma. | oral, |
| Vav3 oncogene is upregulated and a poor prognostic factor in breast cancer patients. | breast, |
| Vav3 proto-oncogene deficiency leads to sympathetic hyperactivity and cardiovascular dysfunction. | None |
| A remarkable new target gene for the dioxin receptor: The Vav3 proto-oncogene links AhR to adhesion and migration. | None |
| Transcriptional factor aryl hydrocarbon receptor (Ahr) controls cardiovascular and respiratory functions by regulating the expression of the Vav3 proto-oncogene. | None |
| Data show that microRNA miR-499-5p targeted 3_ untranslated regions (3_-UTR) of vav 3 oncogene protein (VAV3). | None |
| SH3 domain-dependent interaction of the proto-oncogene product Vav with the focal contact protein zyxin. | None |