| Therefore, our findings indicate that CSF1 signaling is oncogenic during gliomagenesis through a mechanism distinct from modulating glioma-associated microglia/macrophage polarization status | glioma, |
| The uptake of glucose induced by CSF-1 required intact 1-Phosphatidylinositol 3-Kinase and Phospholipase C signalling pathways, as well as the downstream effectors Proto-Oncogene Proteins c-akt and Protein Kinase C. | None |
| Expression pattern and neurotrophic role of the c-fms proto-oncogene M-CSF receptor in rodent Purkinje cells. | None |