| Oncogene-induced sensitization to chemotherapy-induced death requires induction as well as deregulation of E2F1. | None |
| E2F1: a potential negative regulator of hTERT transcription in normal cells upon activation of oncogenic c-Myc. | None |
| E2F-1, a key transcription factor necessary for cell growth, DNA repair, and differentiation, is an attractive target for development of anticancer drugs in tumors that are E2F oncogene addicted. | None |
| The E2F1 oncogene transcriptionally regulates NELL2 in cancer cells. | None |
| oncogenic activities of E2F1 and E2F3 in ErbB2- or Myc-triggered mammary tumorigenesis, and a tumor suppressor role of E2F2 in Myc-mediated mammary tumorigenesis | None |
| Cancerous inhibitor of protein phosphatase 2A contributes to human papillomavirus oncoprotein E7-induced cell proliferation via E2F1. | None |
| These data suggest a model in which cells experiencing oncogene-induced replication stress through deregulation of E2F-dependent transcription. | None |
| Data show that lncRNA-HIT acted as an oncogene through association with E2F transcription factor 1 protein (E2F1). | None |
| Stimulation of E2F1/DP1 transcriptional activity by MDM2 oncoprotein. | None |