| miR-223 functions as a tumor suppresser in osteosarcoma and miR-223/Ect2/p21 signaling is an important pathway that regulates the osteosarcoma cell cycle progression and proliferaion | osteosarcoma, |
| Expression level of ECT2 proto-oncogene correlates with prognosis in glioma patients. | glioma, |
| Ect2 and PKCiota are genetically and functionally linked in NSCLC, acting to coordinately drive tumor cell proliferation and invasion through formation of an oncogenic PKCiota-Par6alpha-Ect2 complex. | lung, |
| Data suggest that ECT2 may play an oncogenic role in the pancreatic ductal adenocarcinoma (PDAC) neoplastic process. | pancreatic, |
| Data suggest that the expression of epithelial cell transforming sequence 2 oncogene (ECT2) can serve as an alternative measurement that can compensate for the inadequacy of the current carcinoembryonic antigen (CEA) test in the diagnosis and monitoring of colorectal cancer patients. | colorectal, |
| Oncogenic Ect2 signaling regulates rRNA synthesis in NSCLC. | lung, |
| Amplification of the Ect2 proto-oncogene and over-expression of Ect2 mRNA and protein in nickel compound and methylcholanthrene-transformed 10T1/2 mouse fibroblast cell lines. | None |
| RB silencing compromises the DNA damage-induced G2/M checkpoint and causes deregulated expression of the ECT2 oncogene. | None |
| Oncogenic activity of Ect2 is regulated through protein kinase C iota-mediated phosphorylation. | None |
| the oncogene ECT2 contributes to epithelial cell reprogramming in idiopathic pulmonary fibrosis | None |
| Chromosomal localization of the human ECT2 proto-oncogene to 3q26.1-->q26.2 by somatic cell analysis and fluorescence in situ hybridization. | None |
| Oncogene ect2 is related to regulators of small GTP-binding proteins. | None |