| Oncogenic signaling from the hematopoietic growth factor receptors c-Kit and Flt3. | None |
| FES kinases are required for oncogenic FLT3 signaling. | None |
| Inhibition of MEK5 by BIX02188 induces apoptosis in cells expressing the oncogenic mutant FLT3-ITD. | None |
| Rho kinase regulates the survival and transformation of cells bearing oncogenic forms of KIT, FLT3, and BCR-ABL. | None |
| Increased binase cytotoxicity toward the cells, expressing FLT3 oncogene, suggests that, as in the case of FDC-P1 cells, transduced by KIT oncogene, the expression of an activated oncogene determines the sensitivity of cells to binase | None |
| [Expression of FLT3-ITD oncogene confers mice progenitor B-cells BAF3 sensitivity to the ribonuclease binase cytotoxic action]. | None |
| Oncogenic roles of PRL-3 in FLT3-ITD induced acute myeloid leukaemia. | None |
| Regulation of Stat5 by FAK and PAK1 in Oncogenic FLT3- and KIT-Driven Leukemogenesis. | None |
| the oncogenic events of FLT3/ITD happen at a cell stage possessing CD123 | None |
| Targeting of SOCS2 by miR-194 resulted in derepression of the oncogenic kinases FLT3 and JAK2, leading to enhanced ERK and STAT3 signaling. | None |
| Overexpression of Abl-related gene tyrosine kinase ABL2 in pro-B cell line Ba/F3 cells expressing an oncogenic mutant of FLT3 (FLT3-ITD) resulted in partial inhibition of FLT3-ITD-dependent cell proliferation. | None |
| LCK cooperates with oncogenic FLT3-ITD in cellular transformation | None |