| Induction of the c-fos proto-oncogene during opiate withdrawal in the locus coeruleus and other regions of rat brain. | brain, |
| [Activation of the c-fos proto-oncogene in different structures of the rat brain during training and pseudoconditioning]. | brain, |
| [Rules of protein expression of proto-oncogene (c-Fos/c-Jun) in different brain areas and nucleus of psychological stressed mice and the regulatory effect of modified Xiaoyao Pill]. | brain, |
| Possible collaboration between c-fos and c-myc proto-oncogene products in in vivo lymphomagenesis. | lymphoma, |
| High levels of c-fos proto-oncogene expression in normal human adult skin. | skin, |
| Stimulation of adrenal medullary cells in vivo and in vitro induces expression of c-fos proto-oncogene. | kidney, |
| Interleukin-1 induces rapid and transient expression of the c-fos proto-oncogene in isolated pancreatic islets and in purified beta-cells. | pancreatic, |
| The proto-oncogene c-fos is over-expressed in the majority of human osteosarcomas. | osteosarcoma, |
| Expression of the fos oncogene in B16 melanoma cells exhibiting different metastatic abilities. | skin, |
| Phorbol ester mimics ACTH action in corticoadrenal cells stimulating steroidogenesis, blocking cell cycle, changing cell shape, and inducing c-fos proto-oncogene expression. | kidney, |
| Application of potassium chloride to the brain surface induces the c-fos proto-oncogene: reversal by MK-801. | brain, |
| N-methyl-D-aspartate receptors mediate activation of the c-fos proto-oncogene in a model of brain injury. | brain, |
| gp130-mediated induction of the immediate early genes FBJ osteosarcoma oncogene and early growth response 1 is also prolonged in STAT3(-/-) cells, with STAT1 binding to their promoters | osteosarcoma, |
| The nuclear import of oncoprotein hepatitis B X-interacting protein depends on interacting with c-Fos and phosphorylation of both proteins in breast cancer cells. | breast, |
| [Effects of intracellular Na+/K+ ratio on expression of c-fos oncogene and beta-actin gene in ascitic cells of leukemia P-388 and Ehrlich tumor]. | leukemia, |
| Topical application of preparations containing DNA repair enzymes prevents ultraviolet-induced telomere shortening and c-FOS proto-oncogene hyperexpression in human skin: an experimental pilot study. | skin, |
| Human osteosarcoma cells respond to sorafenib chemotherapy by downregulation of the tumor progression factors S100A4, CXCR4 and the oncogene FOS. | osteosarcoma, |
| Proto-oncogene c-fos is transiently induced in the rat cerebral cortex after forebrain ischemia. | brain, |
| By targeting the proto-oncogene Fos, miR-101 is involved in G1-to-S phase transition in cervical cancer cells in vitro. | cervical, |
| miR-101 inhibits the G1-to-S phase transition of cervical cancer cells by targeting Fos. | cervical, |