Sentence and cancer type
Showing 101-120 of 227 items.
SentenceCancertype
Intrinsic regulation of the interactions between the SH3 domain of p85 subunit of phosphatidylinositol-3 kinase and the protein network of BCR/ABL oncogenic tyrosine kinase. None
Photodynamic treatment (ALA-PDT) suppresses the expression of the oncogenic Bcr-Abl kinase and affects the cytoskeleton organization in K562 cells. None
Growth, differentiation, and malignant transformation of pre-B cells mediated by inducible activation of v-Abl oncogene. None
Oxidant-induced restriction polymorphism maps to kinase region of c-abl oncogene. None
MUC1 oncoprotein blocks nuclear targeting of c-Abl in the apoptotic response to DNA damage. None
Clonally related IL-7-independent tumors induced by oncogenes abl + myc or raf + myc have reduced sensitivity to TGF-beta1.None
BCR sequences essential for transformation by the BCR-ABL oncogene bind to the ABL SH2 regulatory domain in a non-phosphotyrosine-dependent manner. None
The abl oncogene family and apoptosis. None
Regulation of cellular transformation by oncogenic and normal Abl kinases. None
Expression of the mouse c-abl type IV proto-oncogene product in the insect cell baculovirus system. None
Ubp43 deficiency increases the resistance to oncogenic transformation by BCR-ABLNone
Structural investigation of PAP derivatives by CoMFA and CoMSIA reveals novel insight towards inhibition of Bcr-Abl oncoprotein. None
High levels of the BCR/ABL oncoprotein are required for the MAPK-hnRNP-E2 dependent suppression of C/EBPalpha-driven myeloid differentiation. None
ds-Oligonucleotide-peptide conjugates featuring peptides from the leucine-zipper region of Fos as switchable receptors for the oncoprotein Jun. None
Differential repression of c-myc and cdc2 gene expression by ERF and PE-1/METS. None
ERF is targeted for inactivation by transforming oncogenes such as vAbl.None
Expression of the p210BCR-ABL oncoprotein drives centrosomal hypertrophy and clonal evolution in human U937 cells. None
An enhanced association of RACK1 with Abl in cells transfected with oncogenic ras. None
Identification of novel posttranscriptional targets of the BCR/ABL oncoprotein by ribonomics: requirement of E2F3 for BCR/ABL leukemogenesis. None
These results demonstrate that allele-specific changes in gene expression, with selective, progressive silencing of the wild-type ABL1 allele in favor of the oncogenic BCR-ABL1 allele occur in CML patients with therapy-resistant disease.None