| FUS1 acts as a tumor-suppressor gene by upregulating miR-197 in human glioblastoma | glioblastoma, |
| Characterization of a previously unknown radioprotective function of the mitochondrial tumor suppressor Fus1. | None |
| Mitochondria, calcium, and tumor suppressor Fus1: At the crossroad of cancer, inflammation, and autoimmunity | None |
| Tumor suppressor candidate 2 (TUSC2, FUS-1) and human cancers | None |
| Structure-function studies of the BTB/POZ transcriptional repression domain from the promyelocytic leukemia zinc finger oncoprotein. | leukemia, |
| Involvement of the pro-oncoprotein TLS (translocated in liposarcoma) in nuclear factor-kappa B p65-mediated transcription as a coactivator. | sarcoma, |
| Myxoid liposarcoma FUS-DDIT3 fusion oncogene induces C/EBP beta-mediated interleukin 6 expression. | sarcoma, |
| The myxoid/round cell liposarcoma fusion oncogene FUS-DDIT3 and the normal DDIT3 induce a liposarcoma phenotype in transfected human fibrosarcoma cells. | fibrosarcoma, |
| FUS-DDIT3 is a chimeric oncogene generated by the most common chromosomal translocation t(12;16)(q13;p11) associated to liposarcomas. | sarcoma, |
| Fat-specific FUS-DDIT3-transgenic mice establish PPARgamma inactivation is required to liposarcoma development. | sarcoma, |
| A mechanistic study on the metastasis inducing function of FUS-CHOP fusion protein in liposarcoma. | sarcoma, |
| The herein presented data uncover a novel mechanism by which the fusion oncogene FUS-CHOP actively promotes invasion in myxoid and round cell liposarcoma through the activation of a SRC/FAK/RHO/ROCK signaling axis. | sarcoma, |
| Human translocation liposarcoma-CCAAT/enhancer binding protein (C/EBP) homologous protein (TLS-CHOP) oncoprotein prevents adipocyte differentiation by directly interfering with C/EBPbeta function. | sarcoma, |
| Human POMp75 is identified as the pro-oncoprotein TLS/FUS: both POMp75 and POMp100 DNA homologous pairing activities are associated to cell proliferation. | None |
| Human 75-kDa DNA-pairing protein is identical to the pro-oncoprotein TLS/FUS and is able to promote D-loop formation. | None |
| Proto-oncoprotein TLS/FUS is associated to the nuclear matrix and complexed with splicing factors PTB, SRm160, and SR proteins. | None |
| The oncogenic TLS-ERG fusion protein activates two different sets of genes sharing little similarity when transforms hematopoietic cells and fibroblasts. | None |
| The oncogenic TLS-ERG fusion protein exerts different effects in hematopoietic cells and fibroblasts. | None |
| Beta-catenin interacts with the FUS proto-oncogene product and regulates pre-mRNA splicing. | None |
| Development of lipoblasts and the typical MLS/RCLS capillary network could be an effect of the DDIT3 transcription factor partner of the fusion oncogene. | None |