| Silencing of individual genes by DNA methylation is controlled by HRAS oncogenic signalling pathways, yet the mechanisms responsible for initial target gene suppression are variable. | None |
| Expression of the c-Ha-ras oncogene in mouse NIH 3T3 cells induces resistance to cisplatin. | None |
| Identification of mutagenic site of c-H-ras oncogene damaged by N-acetoxyacetylaminofluorene(AAAF). | None |
| Antisense-fos RNA causes partial reversion of the transformed phenotypes induced by the c-Ha-ras oncogene. | None |
| Ultrastructural cytoskeleton alterations and modification of actin expression in the NIH/3T3 cell line after transformation with Ha-ras-activated oncogene. | None |
| Effects of retinoic acid on NIH3T3 cell transformation by the H-ras oncogene. | None |
| Induction of glutathione content in murine melanocytes after transformation with c-H-ras oncogene. | None |
| Inhibition of myogenesis by the H-ras oncogene: implication of a role for protein kinase C. | None |
| Preferential methylation of the Ha-ras proto-oncogene by methylnitrosourea in rat mammary glands. | None |
| [The c-fos proto-oncogene promotor is not regulated by serum, epidermal growth factor, and phorbol ester in embryonal fibroblasts transformed by E1Aad5+cHa-ras-oncogenes]. | None |
| In vitro transformation of mesenchymal stem cells by oncogenic H-rasVal12. | None |
| Ha-ras oncogene activation in mammary glands of N-methyl-N-nitrosourea-treated rats genetically resistant to mammary adenocarcinogenesis. | None |
| Possible application of human c-Ha-ras proto-oncogene transgenic rats in a medium-term bioassay model for carcinogens. | None |
| The oncoprotein H-RasV12 increases mitochondrial metabolism. | None |
| Oncogenic H-Ras V12 promotes anchorage-independent cytokinesis in human fibroblasts. | None |
| V12 H-Ras oncogenic signaling may contribute to anchorage-independent growth and tumorigenesis by promoting the final cleavage furrow ingression during cytokinesis. | None |
| Hot spot amino acid distribution in Ha-ras oncogene product p21: relationship to guanine binding site. | None |
| Infection with M. arginini made rat and mouse embryo fibroblasts susceptible to transformation with oncogenic H-Ras | None |
| The level of oncogene H-Ras correlates with tumorigenicity and malignancy. | None |
| [An increased frequency of allele A6 of the proto-oncogene HRAS1 in cancer patients]. | None |