| Nox1 mediates oncogenic Ras-induced upregulation of VEGF and angiogenesis by activating Sp1 through Ras-ERK-dependent phosphorylation of Sp1. | None |
| Identification of genes that exhibit increased expression after flat reversion of NIH/3T3 cells transformed by human activated Ha-ras oncogene. | None |
| v-myb transformation of Xeroderma pigmentosum human fibroblasts: overexpression of the c-Ha-ras oncogene in the transformed cells. | None |
| Expression of PTEN is suppressed by the oncogenic H-Ras at its protein and transcript levels as well as by oncogenic K- and N-Ras. | None |
| [Activation of the Ha-ras oncogene in tumors induced in mice by transplacental exposure to 7,12-dimethylbenz(a)anthracene]. | None |
| HTLV-1 Tax protein cooperates with Ras in protecting cells from apoptosis. | None |
| Low incidence of Ha-ras oncogene mutations in human epidermal tumors. | None |
| Ha-ras oncogene-induced Stat3 phosphorylation enhances oncogenicity of the cell. | None |
| Ha-ras overexpression increases phosphorylation of Stat3 at serine-727 and tyrosine-705 in 293T cells and enhances oncogenicity of the cell. | None |
| trend of increased H-ras expression was observed in diabetic compared to normal rats during oncogenesis | None |
| Transformation of NIH3T3 cells by transfection with UV-irradiated human c-Ha-ras-1 proto-oncogene DNA. | None |
| [Frequency and rearrangements of alleles of proto-oncogene c-Ha-ras-1 and their association with development of various malignant tumors in man]. | None |
| Findings show that JNK is necessary for oncogenic H-ras-induced, caspase-independent cell death, and that both PI3K and Rac1 activities are required for JNK activation and cell death. | None |
| Ha-ras oncogene mutations in cell lines derived from rat tracheal implants exposed in vivo to 7,12-dimethylbenz[a]anthracene. | None |
| Modeling the effect of the RB tumor suppressor on disease progression: dependence on oncogene network and cellular context. | None |
| A single cellular event for mutations arising in male germ cells that show a paternal age effect; screening of 30 spermatocytic seminomas for oncogenic mutations in 17 genes identified 2 mutations in FGFR3 and 5 mutations in HRAS. | None |
| Silencing of the Lats2 tumor suppressor overrides a p53-dependent oncogenic stress checkpoint and enables mutant H-Ras-driven cell transformation. | None |
| Impairment of mitochondrial respiration in mouse fibroblasts by oncogenic H-RAS(Q61L). | None |
| Molecular cloning of a rearranged HRAS1 oncogene in chromosome mediated gene transfer associated with elevated tumorigenicity. | None |
| Characterization of recessive (mediator-) revertants from NIH 3T3 cells transformed with a c-H-ras oncogene. | None |