| MNU-induced point mutation and activation of c-Ha-ras oncogene in cell lines derived from human pancreas explants. | None |
| Tetra-amidines exhibiting anti-proteinase activity: effects on oriented migration and in vitro invasiveness of a Chinese hamster cell line transfected with the activated human T24-Ha-ras-1 oncogene. | None |
| NADPH oxidase NOX4 is a critical mediator in oncogenic H-RasV12-induced DNA-damage response and subsequent senescence. | None |
| Our findings suggest that the HRAS mutations identified in Costello syndrome (CS) are sufficient to cause oncogene-induced senescence and that cellular senescence might therefore contribute to the pathogenesis of CS. | None |
| [The effect of Ha-ras oncogene on cells immortalized by the gene for polyomavirus large T-antigen]. | None |
| Immune response to progressor variants derived from transfection of an ultraviolet radiation-induced C3H mouse regressor tumor cell line with activated Harvey-ras oncogene. | None |
| Role of the Ha-ras (RasH) oncogene in mediating progression of the tumor cell phenotype (review). | None |
| Growth advantage by overexpression of normal Harvey ras proto-oncogene in cultured rat epidermal keratinocytes. | None |
| reduced C4ST-1 expression and chondroitin sulfation imbalance mediating the effects of oncogenic HRAS signaling in the pathogenesis of Costello syndrome. | None |
| A critical role of MYC for transformation of human cells by HPV16 E6E7 and oncogenic HRAS. | None |
| Crystal structures of the state 1 conformations of the GTP-bound H-Ras protein and its oncogenic G12V and Q61L mutants. | None |
| The GTP bound state 1 crystal structures of wild-type H-Ras together with the oncogenic G12V and Q61L mutants. | None |
| Methylation pattern of oncogene HRAS gene promoter region and its clinical relevance to urocystic tumorigenesis. | None |
| Interplay between oncogenic K-Ras and wild-type H-Ras in Caco2 cell transformation. | None |
| ASPP2 modulates oncogenic RAS-induced autophagic activity to dictate the cellular response to RAS: to proliferate or senesce. | None |
| Dissecting the different biological effects of oncogenic Ras isoforms in cancer cell lines: could stimulation of oxidative stress be the one more weapon of H-Ras? Regulation of oxidative stress and Ras biological effects. | None |
| Oncogene Ras/phosphatidylinositol 3-kinase signaling targets histone H3 acetylation at lysine 56. | None |
| Cellular redox imbalance and changes of protein S-glutathionylation patterns are associated with senescence induced by oncogenic H-ras. | None |
| we induced tumors in mice via the injection of transposons encoding three oncogenes(HRAS, c-Myc and shp53) and a plasmid-expressing transposase. | None |
| Dynamic palmitoylation links cytosol-membrane shuttling of acyl-protein thioesterase-1 and acyl-protein thioesterase-2 with that of proto-oncogene H-ras product and growth-associated protein-43. | None |