| [Detection and molecular genetic characterization of the c-Ha-ras 1 oncogene in human breast cancer, malignant melanoma and neuroblastoma]. | skin,neuroblastoma,breast, |
| A position 12-activated H-ras oncogene in all HS578T mammary carcinosarcoma cells but not normal mammary cells of the same patient. | sarcoma, |
| Human malignant tumours of the breast, as compared to their respective normal tissue, have elevated expression of the Harvey ras oncogene. | breast, |
| T24 human bladder carcinoma oncogene is an activated form of the normal human homologue of BALB- and Harvey-MSV transforming genes. | bladder, |
| Complete nucleotide sequences of the T24 human bladder carcinoma oncogene and its normal homologue. | bladder, |
| A molecular approach to leukemogenesis: mouse lymphomas contain an activated c-ras oncogene. | lymphoma, |
| Mechanism of activation of an N-ras oncogene of SW-1271 human lung carcinoma cells. | lung, |
| Mouse skin carcinomas induced in vivo by chemical carcinogens have a transforming Harvey-ras oncogene. | skin, |
| Nucleotide sequence analysis of the T24 human bladder carcinoma oncogene. | bladder, |
| Granulocyte-colony stimulating factor (G-CSF) production of human fibroblasts (KMST-6/RAS line) transformed with 60Co gamma rays and c-Ha-ras oncogene. | colorectal, |
| Human fibroblasts (KMST-6/RAS line) transformed with 60Co gamma-rays and c-Ha-ras oncogene constitutively produce a large amount of human granulocyte-colony stimulating factor (G-CSF). | colorectal, |
| Human fibroblasts (KMST-6/RAS cell line) transformed with 60Co gamma-rays and c-Ha-ras oncogene produce a large amount of granulocyte colony-stimulating factor (G-CSF); production is enhanced by cAMP, theophylline, and butyrate. | colorectal, |
| Mutations, expression and genomic instability of the H-ras proto-oncogene in squamous cell carcinomas of the head and neck. | HNSC, |
| H-ras oncogene mutation spectra in B6C3F1 and C57BL/6 mouse liver tumors provide evidence for TCDD promotion of spontaneous and vinyl carbamate-initiated liver cells. | liver, |
| Mutations in the ras proto-oncogene: clues to etiology and molecular pathogenesis of mouse liver tumors. | liver, |
| Direct tumorigenic conversion of human gallbladder carcinoma cells by v-src but not by activated c-H-ras oncogene. | bladder, |
| c-Ha-ras oncogene transfection does not affect NK cell sensitivity of human colorectal carcinoma cell lines. | colorectal, |
| Constitutive expression of the c-H-ras oncogene inhibits doxorubicin-induced apoptosis and promotes cell survival in a rhabdomyosarcoma cell line. | sarcoma, |
| H-ras oncogene mutation and human papillomavirus infection in oral carcinomas. | oral |
| Detection of high incidence of H-RAS oncogene point mutations in acute myelogenous leukemia. | leukemia, |