| Hepatic tumors induced by carbon tetrachloride in transgenic mice carrying a human c-H-ras proto-oncogene without mutations. | liver, |
| Modulation of mdr-1 expression by a H-ras oncogene in a human colon carcinoma cell line. | colorectal, |
| Amplification of c-myc oncogene and absence of c-Ha-ras point mutation in human bone sarcoma. | sarcoma, |
| Membrane association of cathepsin B can be induced by transfection of human breast epithelial cells with c-Ha-ras oncogene. | breast, |
| Allele loss and point mutation in codons 12 and 61 of the c-Ha-ras oncogene in carcinogen-transformed human breast epithelial cells. | breast, |
| Human retinoblastoma gene product prevents c-Ha-ras oncogene mediated cellular transformation of mouse fibroblasts. | retinoblastoma, |
| Structure of a Moloney murine leukemia virus-virus-like 30 recombinant: implications for transduction of the c-Ha-ras proto-oncogene. | leukemia, |
| The structure and function of the H-ras proto-oncogene are not altered in rat liver tumors initiated by 2-acetylaminofluorene, 2-acetylaminophenanthrene and trans-4-acetylaminostilbene. | liver, |
| DNA ploidy, c-myc oncoprotein p62 and v-H-ras oncoprotein p21 in colorectal carcinoma. | colorectal, |
| Ha-ras oncogene expression abrogates a pH dependent endonuclease activity of apoptosis in normal rat kidney cells. | kidney, |
| Suppression of the metastatic phenotype of a mouse skin carcinoma cell line independent of E-cadherin expression and correlated with reduced Ha-ras oncogene products. | skin, |
| Transformation of immortalized woodchuck hepatic cell lines with the c-Ha-ras proto-oncogene. | liver, |
| Phenylacetate inhibits protein isoprenylation and growth of the androgen-independent LNCaP prostate cancer cells transfected with the T24 Ha-ras oncogene. | prostate, |
| C-Ha-ras oncogene in oral leukoplakia tissues. | oral |
| Ha-ras oncogene (codon 12) mutation in thyroid carcinogenesis: analysis of 60 benign and malignant thyroid tumors. | thyroid, |
| Evaluation of c-ras oncogene product (p21) in superficial bladder cancer. | bladder, |
| Glucocorticoid and estrogen receptors have elevated activity in human endometrial and ovarian tumors as compared to the adjacent normal tissues and recognize sequence elements of the H-ras proto-oncogene. | ovarian,endometrial, |
| Alterations of tumor suppressor genes and the H-ras oncogene in oral squamous cell carcinoma. | oral |
| Geranylgeranyl as well as farnesyl moiety is transferred to Ras p21 overproduced in adrenocortical cells transformed by c-Ha-rasEJ oncogene. | adrenocortical, |
| [Restriction polymorphism of the proto-oncogene c-Ha-ras-1 in patients with multiple primary malignant neoplasms and non-small-cell lung cancer]. | neoplasm,lung, |