Sentence and cancer type
Showing 1-20 of 24 items.
SentenceCancertype
Effects of the SV40 large T antigen and EJ ras oncogene on fibronectin localization in human endometrial cells as viewed by confocal laser scanning microscopy. endometrial,
Expression of ERas oncogene in gastric carcinoma. gastric,
Epigenetic regulation of the embryonic oncogene ERas in gastric cancer cells. gastric,
Loss of methylation in the promoter of ERas might be one of mechanisms responsible for the re-expression of an embryonic oncogene in gastric cancer.gastric,
Fluorescent laser scanning microscopy of F-actin disruption in human endometrial stromal cells expressing the SV40 large T antigen and the EJ ras oncogene. endometrial,
Identification of HLA-A*2402-restricted epitope peptide derived from ERas oncogene expressed in human scirrhous gastric cancer. gastric,
Reduced hormone-stimulated adenylate cyclase activity in NIH-3T3 cells expressing the EJ human bladder ras oncogene. bladder,
Development of keratoacanthomas and squamous cell carcinomas in transgenic rabbits with targeted expression of EJras oncogene in epidermis. None
Role of ERas in promoting tumour-like properties in mouse embryonic stem cells. None
transforming oncogene ERas is important in the tumour-like growth properties of embryonic stem cellsNone
Transforming growth factor beta stimulates mitogenically mouse NIH3T3 fibroblasts and those cells transformed by the EJ-H ras oncogene. None
EJ-ras oncogene transfection of endothelial cells upregulates the expression of syndecan-4 and downregulates heparan sulfate sulfotransferases and epimerase. None
ERas oncogene expression and epigenetic regulation by histone acetylation in human cancer cells. None
Inhibition of HSV-1 multiplication in rat embryo fibroblasts constitutively expressing the EJ-ras oncogene. None
Glucocorticoid dexamethasone reversibly complements EJ-RAS oncogene to transform mouse embryo BALB-3T3 cells. None
Tumorigenicity of human mesothelial cell line transfected with EJ-ras oncogene. None
Inhibition of adenylate cyclase and phospholipase A2/C in NIH-3T3 cells expressing the EJ-ras oncogene. None
Loss of platelet-derived growth factor-stimulated phospholipase activity in NIH-3T3 cells expressing the EJ-ras oncogene. None
Normal human mesothelial cells and fibroblasts transfected with the EJras oncogene become EGF-independent, but are not malignantly transformed. None
Platelet-derived growth factor does not induce c-fos in NIH 3T3 cells expressing the EJ-ras oncogene. None