| [Effects of chronic hypoxia on the expression of oncogene jun fos and myb mRNA in rat lung]. | lung, |
| The expression of proto-oncogene c-jun in human pancreatic cancer. | pancreatic, |
| Alteration of jun proto-oncogene status by plasmid transfection affects growth of human ovarian cancer cells. | ovarian, |
| A dominant role for the c-Jun NH2-terminal kinase in oncogenic ras-induced morphologic transformation of human lung carcinoma cells. | lung, |
| Suppression of proto-oncogene (AP-1) in a model of skin epidermal hyperproliferation is reversed by topical application of 13-hydroxyoctadecadienoic acid and 15-hydroxyeicosatrienoic acid. | skin, |
| Activation of oncogenic transcription factor AP-1 in T cells infected with human T cell leukemia virus type 1. | leukemia, |
| Bombesin antagonists inhibit growth of MDA-MB-435 estrogen-independent breast cancers and decrease the expression of the ErbB-2/HER-2 oncoprotein and c-jun and c-fos oncogenes. | breast, |
| Proto-oncogene c-jun and c-fos messenger RNAs increase in the liver of carnitine-deficient juvenile visceral steatosis (jvs) mice. | liver, |
| CCK, bombesin, and carbachol stimulate c-fos, c-jun, and c-myc oncogene expression in rat pancreatic acini. | pancreatic, |
| Upregulation of the matrix metalloproteinase-1 gene by the Ewing s sarcoma associated EWS-ER81 and EWS-Fli-1 oncoproteins, c-Jun and p300. | sarcoma, |
| Vav-promoter regulated oncogenic fusion protein NPM-ALK in transgenic mice causes B-cell lymphomas with hyperactive Jun kinase. | lymphoma, |
| Expression of c-jun oncogene in hyperplastic and carcinomatous human prostate. | prostate, |
| [Rules of protein expression of proto-oncogene (c-Fos/c-Jun) in different brain areas and nucleus of psychological stressed mice and the regulatory effect of modified Xiaoyao Pill]. | brain, |
| JUN oncogene amplification and overexpression block adipocytic differentiation in highly aggressive sarcomas. | sarcoma, |
| Blockade of AP-1 activity by dominant-negative TAM67 can abrogate the oncogenic phenotype in latent membrane protein 1-positive human nasopharyngeal carcinoma. | nasopharyngeal,laryngeal, |
| The AP-1 transcription factor regulates breast cancer cell growth via cyclins and E2F factors. | breast, |
| Novel pathway for thyroid hormone receptor action through interaction with jun and fos oncogene activities. | thyroid, |
| c-Jun amplification and overexpression are oncogenic in liposarcoma but not always sufficient to inhibit the adipocytic differentiation programme. | sarcoma, |
| The identification of c-Jun as a transcriptional regulator of PDK1 expression highlights key mechanisms underlying c-Jun oncogenic activity, and provides new insight into the nature of up-regulated Akt and PKC in melanoma. | skin, |
| Transcription factor levels in medullary thyroid carcinoma cells differentiated by Harvey ras oncogene: c-jun is increased. | thyroid, |