| Induction of proto-oncogene c-jun product in rat liver after partial hepatectomy. | liver, |
| NF-kB and c-Jun induce the expression of the oncogenic miR-221 and miR-222 in prostate carcinoma and glioblastoma cells. | glioblastoma,prostate, |
| C-jun oncogene expression in nonsmall cell lung-cancer. | lung, |
| Human miR-1285-3p could directly repress JUN oncogene expression in hepatocellular carcinoma cells. | liver, |
| miR-1285-3p acts as a potential tumor suppressor miRNA via downregulating JUN expression in hepatocellular carcinoma. | liver, |
| The mucin MUC4 is a transcriptional and post-transcriptional target of K-ras oncogene in pancreatic cancer. Implication of MAPK/AP-1, NF-kappaB and RalB signaling pathways. | pancreatic, |
| The positive feedback regulation of OCT4 and c-JUN, resulting in the continuous expression of oncogenes such as c-JUN, seems to play a critical role in the determination of the cell fate decision from induced pluripotent stem cells to cancer stem cells in liver cancer. | liver, |
| that COP1 may play a role in promoting glioma cell proliferation by interacting with and downregulating tumor suppressor p53 rather than oncogenic protein c-JUN | glioma, |
| this study revealed the functional and mechanistic links between CDK5 and the oncogenic ERK5-AP-1 signaling pathway in the pathogenesis of colorectal cancer. | colorectal, |
| Avian sarcoma virus 17 carries the jun oncogene. | sarcoma, |
| Nlrp12(-/-) mice were highly susceptible to diethylnitrosamine-induced hepatocellular carcinoma with increased inflammation, hepatocyte proliferation, and tumor burden. Consistently, Nlrp12(-/-) tumors showed higher expression of proto-oncogenes cJun and cMyc and downregulation of tumor suppressor p21. | liver, |
| Results suggest possibilities regarding targeting of the mucosa-associated lymphoid tissue lymphoma translocation protein-1 (MALT1)-Jun proto-oncogene (c-Jun)-glutaminase (GLS1) axis as a potential therapeutic strategy against activated B cell-like diffuse large B-cell lymphoma (ABC-DLBCL). | lymphoma, |
| Pronounced activation of protein kinase C, ornithine decarboxylase and c-jun proto-oncogene by paraquat-generated active oxygen species in WI-38 human lung cells. | lung, |
| Lack of correlated expression between the glutathione S-transferase P-form and the oncogene products c-Jun and c-Fos in rat tissues and preneoplastic hepatic foci. | liver, |
| c-jun oncogene expression in transitional cell carcinoma of the urinary bladder. | bladder, |
| Studies of c-jun oncogene expression in human breast using a new monoclonal antibody, NCL-DK4. | breast, |
| Tumorigenic and metastatic properties of two ras-oncogene transfected rat fibrosarcoma cell lines defective in c-jun. | fibrosarcoma, |
| Prognostic significance of the expression of c-fos, c-jun and c-erbB-1 oncogene products in human squamous cell lung carcinomas. | lung, |
| Immunohistochemical detection of RAS, JUN, FOS, and p53 oncoprotein expression in human colorectal adenomas and carcinomas. | colorectal, |
| The Bcr-Abl leukemia oncogene activates Jun kinase and requires Jun for transformation. | leukemia, |