Sentence and cancer type
Showing 41-60 of 214 items.
SentenceCancertype
Transfection of proto oncogene c-jun into a mouse neuroblastoma cell line potentiates differentiation. neuroblastoma,
Pituitary adenylate-cyclase-activating polypeptide stimulates proto-oncogene expression and activates the AP-1 (c-Fos/c-Jun) transcription factor in AR4-2J pancreatic carcinoma cells. pancreatic,pituitary,
Correlated expression of glutathione S-transferase-pi and c-Jun or other oncogene products in human squamous cell carcinomas of the head and neck: relevance to relapse after radiation therapy. HNSC,
Value of glutathione S-transferase pi and the oncogene products c-Jun, c-Fos, c-H-Ras, and c-Myc as a prognostic indicator in endometrial carcinomas. endometrial,
Heparin-binding epidermal growth factor-like growth factor, a v-Jun target gene, induces oncogenic transformation. None
Effects of capsaicin on induction of c-jun proto-oncogene expression in Fisher-344 rats by N-methyl-N -nitro-N-nitrosoguanidine. None
Requirement for Ras/Rac1-mediated p38 and c-Jun N-terminal kinase signaling in Stat3 transcriptional activity induced by the Src oncoprotein. None
Multiple mitogen-activated protein kinase signaling pathways connect the cot oncoprotein to the c-jun promoter and to cellular transformation. None
Glutathione-S-Transferase as a selective inhibitor of oncogenic ras-p21-induced mitogenic signaling through blockade of activation of jun by jun-N-terminal kinase. None
Oncogenic transformation by ras and fos is mediated by c-Jun N-terminal phosphorylation. None
Induction of oocyte maturation by jun-N-terminal kinase (JNK) on the oncogenic ras-p21 pathway is dependent on the raf-MEK signal transduction pathway. None
The evi-1 oncoprotein inhibits c-Jun N-terminal kinase and prevents stress-induced cell death. None
Role of PRKCM (PKCmu) in radiation-induced increase of JUN proto-oncogene mRNA levels in B-lineage lymphoid cells. None
Directed mutation of the basic domain of v-Jun alters DNA binding specificity and abolishes its oncogenic activity in chicken embryo fibroblasts. None
Endogenous AP-1 levels necessary for oncogenic activity are higher than those sufficient to support normal growth. None
Silica-induced activation of c-Jun-NH2-terminal amino kinases, protracted expression of the activator protein-1 proto-oncogene, fra-1, and S-phase alterations are mediated via oxidative stress. None
Jun, the oncoprotein. None
JAC, a direct target of oncogenic transcription factor Jun, is involved in cell transformation and tumorigenesis. None
A genetic analysis of the cytological region 46C-F containing the Drosophila melanogaster homolog of the jun proto-oncogene. None
Okadaic acid stimulates osteopontin expression through de novo induction of AP-1. None