| Epidermal growth factor receptor signaling synergizes with Hedgehog/GLI in oncogenic transformation via activation of the MEK/ERK/JUN pathway. | None |
| Loss of E-cadherin-mediated cell-cell contacts activates a novel mechanism for up-regulation of the proto-oncogene c-Jun. | None |
| v-jun oncogene prevents terminal differentiation and suppresses muscle-specific gene expression in ASV-17-infected muscle cells. | None |
| Involvement of NF-kappaB and AP-1 in COX-2 upregulation by human papillomavirus 16 E5 oncoprotein. | None |
| C-fos and c-jun proto-oncogene expression is decreased in psoriasis: an in situ quantitative analysis. | None |
| The transactivating domain of the c-Jun proto-oncoprotein is required for cotransformation of rat embryo cells. | None |
| The control of tcf4 expression by JNK/c-Jun leads to a positive feedback loop that connects JNK and Wnt signalling regulates the physiological function of progenitor cells and oncogenic transformation. | None |
| Human papillomavirus type 16 oncoprotein E7 suppresses cadherin-mediated cell adhesion via ERK and AP-1 signaling. | None |
| Oncogenic Ras signals through activation of both phosphoinositide 3-kinase and Rac1 to induce c-Jun NH2-terminal kinase-mediated, caspase-independent cell death. | None |
| The cancer/testis antigen CAGE with oncogenic potential stimulates cell proliferation by up-regulating cyclins D1 and E in an AP-1- and E2F-dependent manner. | None |
| Acrylamide can induce high protein expressions of c-fos and c-jun proto-oncogenes and apoptosis in L-02 cells. | None |
| [Effects of acrylamide on the protein expressions of c-fos and c-jun proto-oncogenes in cell L-02]. | None |
| Heterodimerization with Fra-1 cooperates with the ERK pathway to stabilize c-Jun in response to the RAS oncoprotein. | None |
| c-Jun N-terminal kinase/c-Jun inhibits fibroblast proliferation by negatively regulating the levels of stathmin/oncoprotein 18. | None |
| show that STS-26T clones conditionally expressing oncogenic N-Ras show increased phosphorylated ERK1,2 and phosphorylated JNK expression concomitant with increased AP-1 activity | None |
| Different members of the jun proto-oncogene family exhibit distinct patterns of expression in response to type beta transforming growth factor. | None |
| Chromosomal localization of the three members of the jun proto-oncogene family in mouse and man. | None |
| Increased intracellular Ca2+ is necessary for maximal expression of the proto-oncogene c-jun in the Jurkat T-cell line. | None |
| Estrogen induced expression of the C-jun proto-oncogene in the immature and mature rat uterus. | None |
| Induction of the proto-oncogene c-jun by angiotensin II. | None |