| NANOG upregulates c-Jun oncogene expression through binding the c-Jun promoter. | None |
| Results suggest that proto-oncogene protiins c-Jun and mitogen-activated protein 3 kinase 1 (MAP3K1) represent parallel pathways in the control of eyelid closure. | None |
| Nerve growth factor induces the proto-oncogene c-jun in PC12 cells. | None |
| Peptide-DNA conjugates as tailored bivalent binders of the oncoprotein c-Jun. | None |
| HP1a/KDM4A is involved in the autoregulatory loop of the oncogene gene c-Jun. | None |
| Data indicate proto-oncogene c-Jun as a guardian of somatic cell fate and its suppression opens the gate to pluripotency. | None |
| Genome-wide association between YAP/TAZ/TEAD and AP-1 at enhancers drives oncogenic growth. | None |
| Human immunodeficiency virus long terminal repeat responds to transformation by the mutant T24 H-ras1 oncogene and it contains multiple AP-1 binding TPA-inducible consensus sequence elements. | None |
| Human proto-oncogene c-jun encodes a DNA binding protein with structural and functional properties of transcription factor AP-1. | None |
| Data suggest that Sf1 and c-jun interact and cooperate to activate the Fdx1 promoter in MA-10 (tumorigenic cell line) and TM3 (non-tumorigenic cell line) Leydig cells; such activation requires different regulatory elements located between -124 and -306 bp of Fdx1 promoter and involves recruitment of Sf1 to this region. (Sf1 = splicing factor 1; c-jun = proto-oncogene c-jun; Fdx1 = ferredoxin 1) | None |
| Macrophage-associated lipin-1 contributes to atherogenesis through persistent activation of a protein kinase Calpha/betaII-extracellular receptor kinase1/2-jun proto-oncogene signaling cascade that contributes to foam cell proinflammatory responses. | None |
| The cross-pathway control gene of Neurospora crassa, cpc-1, encodes a protein similar to GCN4 of yeast and the DNA-binding domain of the oncogene v-jun-encoded protein. | None |
| Igf2, a maternal imprinted gene, and c-jun, a proto-oncogene, are regulated by p53 in these cells. DNA damage leads to an increase in p53 abundance in human amniotic fluid cells and to transcriptional activation of its target genes. Interestingly, cell differentiation toward the neural lineage leads to p53 induction as differentiation progresses. | None |
| The DNA-binding domains of the jun oncoprotein and the yeast GCN4 transcriptional activator protein are functionally homologous. | None |
| The JUN oncoprotein, a vertebrate transcription factor, activates transcription in yeast. | None |
| Fos-associated protein p39 is the product of the jun proto-oncogene. | None |
| Epidermal growth factor stimulates transcription of the c-jun proto-oncogene in rat fibroblasts. | None |
| The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1. | None |
| Transcriptional activation by yeast GCN4, a functional homolog to the jun oncoprotein. | None |
| Oncogene jun encodes a sequence-specific trans-activator similar to AP-1. | None |