| Immunohistochemical expression of the c-kit proto-oncogene product in human malignant and non-malignant breast tissues. | breast, |
| Proto-oncogene c-kit expression in malignant melanoma: protein loss with tumor progression. | skin, |
| Assignment of the horse mitochondrial glutamate oxaloacetate transaminase 2 (GOT2) and v-kit Hardy-Zuckerman 4 feline sarcoma viral oncogene homolog (KIT) to horse chromosome 3 by in situ hybridization. | sarcoma, |
| Expression of the c-kit proto-oncogene in rat hepatic allografts during acute rejection. | liver, |
| Spontaneous canine mast cell tumors express tandem duplications in the proto-oncogene c-kit. | None |
| c-kit proto-oncogene exon 8 in-frame deletion plus insertion mutations in acute myeloid leukaemia. | None |
| Regulation of ferritin mRNA translation in primary erythroblasts: exogenous c-Kit plus EpoR signaling mimics v-ErbA oncoprotein activity. | None |
| Three novel mutations of the proto-oncogene KIT cause human piebaldism. | None |
| Gastrointestinal stromal tumor with a novel mutation of KIT proto-oncogene. | None |
| Characterization of an undifferentiated malignancy as a mast cell tumor using mutation analysis in the proto-oncogene c-KIT. | None |
| STI571 inactivation of the gastrointestinal stromal tumor c-KIT oncoprotein: biological and clinical implications. | None |
| The Genomic Structure of the Proto-Oncogene c-kit Encoded at the Murine White Spotting Locus. | None |
| [Expression and clinical significance of c-kit oncogene in gastrointestinal stromal tumors]. | None |
| Human piebaldism: six novel mutations of the proto-oncogene KIT. | None |
| Necessity of tyrosine 719 and PI3-K-mediated signal pathway in constitutive activation and oncogenic potential of c-kit receptor tyrosine kinase with the Asp814Val mutation. | None |
| Necessity of tyrosine 719 and phosphatidylinositol 3 -kinase-mediated signal pathway in constitutive activation and oncogenic potential of c-kit receptor tyrosine kinase with the Asp814Val mutation. | None |
| Functional and phenotypic studies of two variants of a human mast cell line with a distinct set of mutations in the c-kit proto-oncogene. | None |
| KIT and PDGFRA mutations appear to be alternative and mutually exclusive oncogenic mechanisms in gastrointestinal stromal tumors | None |
| Kit kinase is autoinhibited through an intramolecular interaction with the juxtamembrane domain, and tyrosine phosphorylation and oncogenic mutations relieved the regulatory function of the juxtamembrane domain | None |
| Organization and nucleotide sequence of the human KIT (mast/stem cell growth factor receptor) proto-oncogene. | None |