| Expression of IkappaB kinase frees the oncogenic functions of Ras and results in invasive carcinoma resembling squamous cell carcinoma. | None |
| Opposing roles of Ras/Raf oncogenes and the MEK1/ERK signaling module in regulation of expression and adhesive function of this enzyme. | None |
| [Conformational aspects of biological activity of functional fragments of the p21ras oncoproteins. 3. Fragment 34-46 of the native oncoprotein and the decapeptide corresponding to its 35-44 sequence]. | None |
| Tumor induction by an endogenous K-ras oncogene is highly dependent on cellular context. | None |
| Stimulation of mitogen-activated protein kinase by oncogenic Ras p21 in Xenopus oocytes. Requirement for Ras p21-GTPase-activating protein interaction. | None |
| Detection of cellular ki-ras oncogene and Epstein-Barr viral DNA by polymerase chain reaction in situ. | None |
| Activation of extracellular signal-regulated kinase, ERK2, by p21ras oncoprotein. | None |
| Ras oncogene directs expression of a differentially sialylated, functionally altered beta1 integrin. | None |
| Evidence that oocyte maturation induced by an oncogenic ras-p21 protein and insulin is mediated by overlapping yet distinct mechanisms. | None |
| IRF-1 expression became constitutive in ras-transformed NIH 3T3 cells and in cells transformed by oncogenes ets, fes, fos, her-2/neu, met, mos, raf, or trk, suggesting that deregulated IRF-1 expression may be associated with loss of growth control | None |
| Oncogenic RAS is sufficient to initiate myeloid leukemogenesis in mice, and this provides an in vivo system for biologic and preclinical studies | None |
| p21(WAF1/CIP1) at the switch between the anti-oncogenic and oncogenic faces of TGFbeta. | None |
| Conditional expression of oncogenic K-ras from its endogenous promoter induces a myeloproliferative disease. | None |
| [Conformational aspects of the biological action of functional fragments of the p21ras oncoprotein family. 1. Fragments 1-11 and 9-16 of the polypeptide chain]. | None |
| [Conformational aspects of the biological effect of functional fragments of the p21ras oncoprotein family. 2. Fragment 1-16, various sequences]. | None |
| NIH3T3 transfectant containing human K-ras oncogene shows enhanced metastatic activity after in vivo tumor growth or co-culture with fibroblasts. | None |
| Endogenous oncogenic K-ras(G12D) stimulates proliferation and widespread neoplastic and developmental defects. | None |
| Oncogenic ras represses RECK expression via a histone deacetylation mechanism. | None |
| Oncogenic activation of the Ras pathway in murine fibroblasts converts p53 into a senescence inducer through a p21waf1-independent mechanism. | None |
| In contrast to p53 shRNAs, PUMA shRNAs were unable to cooperate with oncogenic ras in transformation | None |