| The dual-specificity kinases, TOPK and DYRK1A, are critical for oocyte maturation induced by wild-type--but not by oncogenic--ras-p21 protein. | None |
| Modulation of tumor induction and progression of oncogenic K-ras-positive tumors in the presence of TGF- b1 haploinsufficiency. | None |
| Three-dimensional structure of p21 in the active conformation and analysis of an oncogenic mutant. | None |
| A specific requirement for Rac1 function in cells expressing oncogenic K-ras. | None |
| Eliminating HSF1 protects mice from tumors induced by mutations of the RAS oncogene or a hot spot mutation in the tumor suppressor p53. In cell culture, HSF1 supports malignant transformation. | None |
| Amplification and activation of c-Ki-ras oncogene in cell line developed from human pancreas explants. | None |
| Detection of Ki-ras proto-oncogene protein by a specific monoclonal antibody. | None |
| Site-specific phosphorylation of raf in cells containing oncogenic ras-p21 is likely mediated by jun-N-terminal kinase. | None |
| activation of the PI3K-AKT-eNOS-(wild-type) Ras pathway by oncogenic Ras in cancer cells is required to initiate and maintain tumour growth | None |
| hRAD17 delayed growth of NIH3T3 fibroblasts transformed by the H-ras oncogene in nude mice. | None |
| Our data suggest the induction of PSA-NCAM by oncogenic K-Ras as a novel molecular mechanism by which E-cadherin-mediated cellular adhesion is reduced and dissemination of tumor cells is facilitated. | None |
| Before panitumumad administration KRAS oncogene must be analysed to determine continuous activation or wild type gene is involved. | None |
| upregulation of transcription is caused by oncogenic Ras signaling through MEK-ERK-dependent phosphorylation of GATA-6 | None |
| Oncogene proteins as biomarkers in the molecular epidemiology of occupational carcinogenesis. The example of the ras oncogene-encoded p21 protein. | None |
| Differential interference of chlorpromazine with the membrane interactions of oncogenic K-Ras and its effects on cell growth. | None |
| Differential interference of chlorpromazine with the membrane interactions of oncogenic K-Ras and its effects on cell growth. | None |
| Intrinsic differences between the catalytic properties of the oncogenic NUP214-ABL1 and BCR-ABL1 fusion protein kinases. | None |
| Transformation by oncogenic Ras expands the early genomic response to transforming growth factor beta in intestinal epithelial cells. | None |
| [The effect of amino acid substitutions on the 3-dimensional structure of fragment 1--16 of the oncoprotein p21 ras]. | None |
| functional link between Stat1 and p27 Kip1, which act in coordination to suppress the oncogenic properties of activated Ras | None |