| A radioimmunocytological quantitative method for the rapid detection of ras oncogene p21 protein in mammalian cells. | None |
| Reactivity of a sulfhydryl group of the ras oncogene product p21 modulated by GTP binding. | None |
| A human Ki-ras oncogene encodes two transforming p21 proteins. | None |
| Establishment of four mouse hybridoma cell lines producing monoclonal antibodies reactive with ras oncogene product p21. | None |
| Activated Ki-ras oncogene in human prostatic adenocarcinoma. | None |
| Visualization of c-Ki-ras-2 oncogene sequences in human pancreas, a chemically induced transplantable carcinoma, and carcinomas of pancreas by in situ hybridization. | None |
| Activation of the c-K-ras oncogene in a human pancreas carcinoma. | None |
| A model for the tertiary structure of p21, the product of the ras oncogene. | None |
| The ras oncogene product p21 is not a regulatory component of adenylate cyclase. | None |
| Four human carcinoma cell lines with novel mutations in position 12 of c-K-ras oncogene. | None |
| Regional mapping of cKi-ras proto-oncogene on mouse chromosome 6 by in situ hybridization. | None |
| Transfection of a rat cell line with the v-Ki-ras oncogene is associated with enhanced susceptibility to natural killer cell lysis. | None |
| Activation of a human c-K-ras oncogene. | None |
| Intrinsic GTPase activity distinguishes normal and oncogenic ras p21 molecules. | None |
| Structure and organization of the human Ki-ras proto-oncogene and a related processed pseudogene. | None |
| Human c-Ki-ras2 proto-oncogene on chromosome 12. | None |
| Acquisition of transforming properties by alternative point mutations within c-bas/has human proto-oncogene. | None |
| X-ray crystal structure analysis of the catalytic domain of the oncogene product p21H-ras complexed with caged GTP and mant dGppNHp. | None |
| p210 bcr-abl confers overexpression of inosine monophosphate dehydrogenase: an intrinsic pathway to drug resistance mediated by oncogene. | None |
| p21ras: an oncoprotein functioning in growth factor-induced signal transduction. | None |