| The findings reveal a tumor suppressive role of hsa-miR-196a2 through regulation of TP63 by ERalpha and/or ERK2 signaling. | None |
| Findings suggest that miR-196a-2 might have a potentially oncogenic role in breast tumorigenesis, and the functional genetic variant in its mature region could serve as a novel biomarker for breast cancer susceptibility: [miR-196a-2] | breast, |
| MiR-196a exerts its oncogenic effect in glioblastoma multiforme by inhibiting IkappaBalpha both in vitro and in vivo. | glioblastoma, |
| the present study showed that miR-196a promoted cell proliferation by targeting UBE2C in breast cancer. Thus, miR-196a may be a potential oncogene in breast cancer and a promising therapeutic target in breast cancer treatment. | breast, |