| miR-205-3p Functions as a Tumor Suppressor in Ovarian Carcinoma | ovarian, |
| EMT was epigenetically driven by chromatin remodeling through H3K27me3 enrichment and later by ensuing DNA methylation to sustain silencing of tumor-suppressive microRNAs (miRNA), miR-200b, miR-200c, and miR-205. | None |
| These results imply that miR-205 is an ESCC-specific miR that exerts tumor-suppressive activities with epithelial mesenchymal transition inhibition by targeting ZEB2. | None |
| MiR-205 is an epigenetically regulated tumor suppressor that targets MED1. | None |
| HBx was able to abrogate the effect of miR-205 on tumor suppression. | None |
| MiR-205, acting as an oncogenic miRNA, may promote the clinical progression of epithelial ovarian cancer patients and enhance the cellular motility in vitro by directly and negatively regulating ZEB1. | ovarian, |
| miR-205 functions as an oncogenic miRNA by directly binding to SMAD4 and PTEN, providing a novel target for the molecular treatment of ovarian cancer. | ovarian, |
| MiR-205 regulates ICT1 in gastric cancer cells and functions as an oncogene and prognostic biomarker. | gastric, |
| findings suggest that miR-205-5p upregulation contributes to MDS by suppressing PTEN and that miR-205-5p thus acts as an oncogene in hematopoietic cells. | None |