| CREB promotes gliomagenesis and acts as a modulator of oncogenic mir-23a, which represses the tumor suppressor PTEN. | glioma, |
| our data provide compelling evidence that miR-23a functions as a tumor suppressor in osteosarcoma, and its inhibitory effect on tumor are mediated chiefly through downregulation of SATB1. | osteosarcoma, |
| MiR-23a regulates TGF-beta-induced epithelial-mesenchymal transition by targeting E-cadherin in lung cancer cells. | lung, |
| CREB promotes gliomagenesis and acts as a modulator of oncogenic mir-23a, which represses the tumor suppressor PTEN. | glioma, |
| cAMP response element-binding protein promotes gliomagenesis by modulating the expression of oncogenic microRNA-23a. | glioma, |
| miR-23a may have an oncogenic function and enhance breast cancer progression. | breast, |
| This study suggested that miR-23a, acting as an oncogenic regulator by directly targeting APAF1 in pancreatic cancer, is a useful potential biomarker in diagnosis and treatment of pancreatic cancer. | pancreatic, |
| miR-23a-5p functions as an oncogene in bladder cancer by affecting cell proliferation, migration and apoptosis. | bladder, |
| High miR-23a expression is the independent prognostic factor of overall and recurrence-free survival rates, and miR-23a may be involved in the onset of hepatocarcinoma as an oncogene. | None |