| MicroRNA-93-5p expression in tumor tissue and its tumor suppressor function via targeting programmed death ligand-1 in colorectal cancer | colorectal, |
| MicroRNA-93 acts as an anti-inflammatory tumor suppressor in glioblastoma | glioblastoma, |
| Functional links between clustered microRNAs: suppression of cell-cycle inhibitors by microRNA clusters in gastric cancer. | gastric, |
| Role of the miR-106b-25 microRNA cluster in hepatocellular carcinoma. | liver, |
| The miR-106b-25 cluster targets Smad7, activates TGF-beta signaling, and induces EMT and tumor initiating cell characteristics downstream of Six1 in human breast cancer. | breast, |
| overexpression of miR-93 in lung cancer cells promoted TGF-b-induced EMT through downregulation of NEDD4L. The analysis of publicly available gene expression array datasets indicates that low NEDD4L expression correlates with poor outcomes among patients with lung cancer, further supporting the oncogenic role of miR-93 in lung tumorigenesis and metastasis. | lung, |
| miR-93 may function as an oncogenic factor in hepatocellular carcinoma, and promotes HCC cell proliferation by targeting PDCD4 | liver, |
| MiR-93-5p is an important oncogene in prostate adenocarcinoma. MiR-93-5p is upregulated in prostate cancer. | prostate, |
| MicroRNA miR-93 promotes tumor growth and angiogenesis by targeting integrin-beta8. | None |