| miR-99a suppresses HCC growth by inducing cell cycle arrest, suggesting miR-99a as potential tumor suppressor for HCC therapeutics. | liver, |
| miR-99a functions as a tumor metastasis suppressor in Oral squamous cell carcinoma cells and mutually regulates IGF1R | oral, |
| Our data indicated that miR-99a might act as a tumor suppressor in bladder cancer and was significantly down-regulated in development of bladder cancer. | bladder, |
| miR-99a/mTOR pathway was involved in diaporine A-induced tumor suppression in non-small cell lung cancer cells. | lung, |
| Tumor-suppressive miR-99a inhibits cell proliferation via targeting of TNFAIP8 in osteosarcoma cells | osteosarcoma, |
| Serum miR-99a expression was down-regulated in breast cancer patients; this down-regulation was associated with poor prognosis, suggesting that serum miR-99a could function as a tumor suppressor in breast cancer. | breast, |
| miR-99a-5p acts as tumor suppressor via targeting to mTOR and enhances RAD001-induced apoptosis in human urinary bladder urothelial carcinoma cells | bladder, |
| Potential tumor?suppressive role of microRNA?99a?3p in sunitinib?resistant renal cell carcinoma cells through the regulation of RRM2 | kidney, |
| miR-99a functions as a tumor suppressor by targeting mTOR. | None |
| Data indicate that proto-oncogene c-Akt (AKT1) is directly involved in miR-99a-mediated tumor suppression. | None |
| MiR-99a functions as a tumor suppressor. | None |