| Acute myeloid leukemia with t(9;11)(p21-22;q23): common properties of dysregulated ras pathway signaling and genomic progression characterize de novo and therapy-related cases. | leukemia, |
| Identification of genes that define transcription factor networks and important genetic pathways acting during progression of leukemia induced by MLL fusion oncogenes. | leukemia, |
| Abrogation of Rac1 signaling causes DNA double-strand breaks in acute monocytic leukemia cells harbouring the MLL-AF9 oncogene. | leukemia, |
| Ras activation seems to complement the MLLT3-MLL oncogene in transformation. | None |
| IGF signaling contributes to malignant transformation of hematopoietic progenitors by the MLL-AF9 oncoprotein. | None |
| Rac1 signaling protects monocytic AML cells expressing the MLL-AF9 oncogene from caspase-mediated apoptotic death. | None |