| 25 years of the c-Myc oncogene. | None |
| The c-myc proto-oncogene regulates cardiac development in transgenic mice. | None |
| Clonally related IL-7-independent tumors induced by oncogenes abl + myc or raf + myc have reduced sensitivity to TGF-beta1. | None |
| complete regression of tumor mass driven by inactivation of the MYC oncogene requires the p53-dependent induction of thrombospondin-1 and the shut down of angiogenesis | None |
| Antiproliferative effects of antisense oligonucleotides directed to the RNA of c-myc oncogene. | None |
| Astrocyte elevated gene-1 (AEG-1) is a target gene of oncogenic Ha-ras requiring phosphatidylinositol 3-kinase and c-Myc. | None |
| DNA repair in the c-myc proto-oncogene locus: possible involvement in susceptibility or resistance to plasmacytoma induction in BALB/c mice. | None |
| identify c-MYC as an essential mediator of NOTCH1 signaling and integrate NOTCH1 activation with oncogenic signaling pathways upstream of c-MYC | None |
| The c-Myc oncogene expression was very low in normal and cancer tissues but highly increased in papillomatosis. | None |
| [Amplification of L-myc oncogene in malignant meningioma. Case report]. | None |
| Histological detection of c-myb and c-myc proto-oncogene expression in infiltrating cells in cutaneous lupus erythematosus-like lesions of MRL/l mice by in situ hybridization. | None |
| oncogenic deregulation of c-MYC prevents repair of replication-stress induced DNA lesions in G(2)-phase; suggests c-MYC-mediated persistence of DNA lesions during mitosis leads to chromosomal missegregation & underlies c-MYC-induced chromosome instability | None |
| Induction of apoptosis and autophagic cell death by the vanillin derivative 6-bromine-5-hydroxy-4-methoxybenzaldehyde is accompanied by the cleavage of DNA-PKcs and rapid destruction of c-Myc oncoprotein in HepG2 cells. | None |
| Expression of exogenous c-myc oncogene does not initiate DNA synthesis in primary rat hepatocyte cultures. | None |
| Stabilization of G-quadruplex DNA and down-regulation of oncogene c-myc by quindoline derivatives. | None |
| Investigation of nuclear c-MYC oncoprotein expression in human hematopoiesis: suitability of a rapid and reliable semiquantitative evaluation system. | None |
| Metabolic enzymes regulated by the Myc oncogene are possible targets for chemotherapy or chemoprevention. | None |
| Mnt takes control as key regulator of the myc/max/mxd network. | None |
| Myc is the most frequently deregulated oncogene in human tumors. Myc function relies on overcoming transcriptional repression by Mnt. Relief of Mnt-mediated repression is more important for target gene regulation than the sole activation by Myc. Review. | None |
| observations raise the possibility that unscheduled replication origin activation at inserted HPV-18 viral DNA sequences triggers DNA amplification in this cancer cell line and the subsequent overexpression of the MYC oncogene | None |