| Results describe for the very first time a direct regulation of an oncogene, MYCN, by a proneural gene, Mash1. | None |
| Increasing extracellular matrix stiffness also reduces expression of N-Myc, a transcription factor involved in multiple aspects of oncogenic proliferation. | None |
| Demonstrate context-dependent transformation of neural stem cells in response to common oncogenic signal N-MYC. | None |
| a general mechanism for p53 inactivation in some embryonal cell types and consequent susceptibility to MycN oncogenesis at the point of embryonal tumor initiation | None |
| analysis of genes interacting functionally with MYCN required to promote fitness of tumor cells facing oncogenic stress | None |
| LIN28B as an important effector of the MYCN oncogenic phenotype and underline the importance of MYCN-regulated miRNAs in establishing the MYCN-driven oncogenic process. | None |
| Oncogenic MYCN re-wires its own interaction and signalling networks to repress regulators of normal cellular developmental processes, such as differentiation. | None |
| Authors identified the transferrin receptor-1 (CD71) to be differentially expressed in neural stem cells and differentiated neurons. In this context, Authors describe a role for N-Myc proto-oncogene (MYCN) in maintaining CD71 expression in proliferating neural cells. | None |
| High resolution mapping of the MYCN proto-oncogene at human chromosome 2p24.3 by fluorescence in situ hybridisation. | None |
| Assignment of human MYCN proto-oncogene to chromosome band 12q24 in higher primates. | None |