| Low incidence of H-, K- and N-ras oncogene mutations in cytological specimens of laryngeal tumours. | laryngeal, |
| The influence of the oncogene N-ras on cell cycle delay in a human melanoma cell line with reduced radioresistance. | skin, |
| Immunohistochemical Expression of N-ras Oncogene is a Late Event in Head and Neck Carcinomas. | HNSC, |
| [Point mutations of N-ras oncogene and abnormal expression of rasp21, p53 proteins in orbital rhabdomyosarcoma]. | sarcoma, |
| Regulation of Fas-mediated apoptosis by N-ras in melanoma. | skin, |
| Suppression of oncogenic NRAS by RNA interference induces apoptosis of human melanoma cells. | skin, |
| oncogenic NRAS is important for avoidance of apoptosis in melanomas that harbor the codon 61 NRAS mutation | skin, |
| Thyroid targeting of the N-ras(Gln61Lys) oncogene in transgenic mice results in follicular tumors that progress to poorly differentiated carcinomas. | thyroid, |
| Mutational activation of the N-ras oncogene assessed in primary clonogenic culture of acute myeloid leukemia (AML): implications for the role of N-ras mutation in AML pathogenesis. | leukemia, |
| Nras gene mutation might be one of the mechanisms of oncogenesis of lung adenocarcinoma. | lung, |
| Differential effects of oncogenic K-Ras and N-Ras on proliferation, differentiation and tumor progression in the colon. | colorectal, |
| Identification of a novel subgroup of melanomas with KIT/cyclin-dependent kinase-4 overexpression. | skin, |
| Oncogenic NRAS has multiple effects on the malignant phenotype of human melanoma cells cultured in vitro. | skin, |
| suppression of oncogenic NRAS by siRNA can induce growth arrest and inhibit invasion of human melanoma cells by modulating the levels of these gene products | skin, |
| NRAS oncogene suppression induces apoptosis followed by more differentiated and less myelosuppressive disease upon relapse of acute myeloid leukemia | leukemia, |
| RAS oncogene suppression induces apoptosis followed by more differentiated and less myelosuppressive disease upon relapse of acute myeloid leukemia. | leukemia, |
| Mutations in the Kirsten-ras oncogene are common but lack correlation with prognosis and tumor stage in human pancreatic carcinoma. | pancreatic, |
| Co-expression of N-ras p21 and C-erbB-2 (neu) oncogene products by common ALL antigen-positive aggressive diffuse lymphoma. | lymphoma, |
| Although oncogenic NRAS expression alone was found to be insufficient to promote tumor formation, loss of functional p53 was found to collaborate with NRAS expression in the genesis of melanoma. | skin, |
| Oncogenic NRAS cooperates with p53 loss to generate melanoma in zebrafish. | skin, |