Sentence and cancer type
Showing 81-100 of 151 items.
SentenceCancertype
Oncogenic signaling of class I PI3K isoforms. None
Comprehensive analysis of oncogenic effects of PIK3CA mutations in human mammary epithelial cells. None
The structure of a human p110alpha/p85alpha complex elucidates the effects of oncogenic PI3Kalpha mutations. None
study reports a 3.0 angstrom resolution structure of a complex between p110alpha and a polypeptide containing the p110alpha-binding domains of p85alpha; results suggest specific mechanisms for the effect of oncogenic mutations in p110alpha and p85alphaNone
structure of the complex between the catalytic subunit of PI3Kalpha, p110alpha, & a portion of its regulatory subunit, p85alpha reveals the majority of the oncogenic mutations occur at the interfaces between p110 domains & between p110 & p85 domainsNone
Insights into the oncogenic effects of PIK3CA mutations from the structure of p110alpha/p85alpha. None
Discovery of drug-resistant and drug-sensitizing mutations in the oncogenic PI3K isoform p110 alpha. None
Class I PI3K in oncogenic cellular transformation. None
PIK3CA cooperates with other phosphatidylinositol 3 -kinase pathway mutations to effect oncogenic transformation. None
Oncogenic HRAS mutations cause prolonged PI3K signaling in response to epidermal growth factor in fibroblasts of patients with Costello syndrome. None
Oncogenic H-Ras and PI3K signaling can inhibit E-cadherin-dependent apoptosis and promote cell survival after photodynamic therapy in mouse keratinocytes. None
Amplifications of 3q26.32 harboring the oncogene PIK3CA were associated with poor prognosis and segregated with the aggressive transcriptional cluster.None
Blockade of Cripto binding to cell surface GRP78 inhibits oncogenic Cripto signaling via MAPK/PI3K and Smad2/3 pathways. None
The transcriptional induction of PIK3CA in tumor cells is dependent on the oncoprotein Y-box binding protein-1. None
AKT-independent signaling downstream of oncogenic PIK3CA mutations in human cancer. None
Oncogene mutations, copy number gains and mutant allele specific imbalance (MASI) frequently occur together in tumor cells. None
Activating mutations in TOR are in similar structures as oncogenic mutations in PI3KCalpha. None
WJD008, a dual phosphatidylinositol 3-kinase (PI3K)/mammalian target of rapamycin inhibitor, prevents PI3K signaling and inhibits the proliferation of transformed cells with oncogenic PI3K mutant. None
Oncogenic mutations of PIK3CA in human cancers. None
The PI3K-Akt mediates oncogenic Met-induced centrosome amplification and chromosome instability. None