| MicroRNA-194 inhibits epithelial to mesenchymal transition of endometrial cancer cells by targeting oncogene BMI-1. | endometrial, |
| MicroRNA-194 inhibits epithelial to mesenchymal transition of endometrial cancer cells by targeting oncogene BMI-1. | endometrial, |
| Akt-mediated phosphorylation of Bmi1 modulates its oncogenic potential, E3 ligase activity, and DNA damage repair activity in mouse prostate cancer. | prostate, |
| The oncoprotein and stem cell renewal factor BMI1 associates with poor clinical outcome in oesophageal cancer patients undergoing preoperative chemoradiotherapy. | esophageal, |
| MicroRNA-218 inhibits cell cycle progression and promotes apoptosis in colon cancer by downregulating BMI1 polycomb ring finger oncogene. | colorectal, |
| Re: akt-mediated phosphorylation of Bmi1 modulates its oncogenic potential, e3 ligase activity, and DNA damage repair activity in mouse prostate cancer. | prostate, |
| BMI-1 might render important oncogenic property of retinoblastomas. | retinoblastoma, |
| The role of BMI1 as a biomarker of cancer stem cells in head and neck cancer: a review. | HNSC, |
| Bmi1 serves as a key driver and biomarker with multiple oncogenic functions underlying tongue tumorigenesis. | tongue, |
| Oncogenic roles of Bmi1 and its therapeutic inhibition by histone deacetylase inhibitor in tongue cancer. | tongue, |
| Bmi-1 is essential for the oncogenic potential in CD133(+) human laryngeal cancer cells. | laryngeal, |
| MicroRNA128a, BMI1 polycomb ring finger oncogene, and reactive oxygen species inhibit the growth of U87 MG glioblastoma cells following exposure to Xray radiation. | glioblastoma, |
| Bmi1 is a potential driver oncogene of bladder cancer, and it may become a potential treatment target for human bladder cancer. | bladder, |
| Data show that the expression of BMI1 polycomb ring finger oncogene protein (Bmi-1) mRNA in the breast cancer tissues was higher than that in the adjacent noncancerous breast tissues. | breast, |
| Data show that C/EBPalpha is a tumor suppressor in lung cancer and that BMI1 is required for the oncogenic process downstream of C/EBPalpha loss. | lung, |
| The Bmi-1 oncogene induces telomerase activity and immortalizes human mammary epithelial cells. | None |
| The Bmi-1 oncogene induces telomerase activity and immortalizes human mammary epithelial cells. | None |
| CALM-AF10+ T-ALL associatd with elevated expression of subset of HOXA genes and some of there transcriptional and functional regulators, as well as specific overexpression of oncogene BMI1 | None |
| CALM-AF10+ T-ALL expression profiles are characterized by overexpression of HOXA and BMI1 oncogenes. | None |
| Study shows that the ability of the oncogene BMI1 to repress the INK4A-ARF locus requires its direct association and is dependent on the continued presence of the EZH2-containing Polycomb-Repressive Complex 2 complex. | None |