| snoN protein has both oncogenic and tumor suppressive properties in colorectal tumorigenesis. | colorectal, |
| SnoN functions as a tumour suppressor by inducing premature senescence. | None |
| These data strongly suggest that SnoN can function as a tumor suppressor at early stages of tumorigenesis in human cancer tissues. | None |
| snoN protein has both oncogenic and tumor suppressive properties in colorectal tumorigenesis. | colorectal, |
| Data show that dominant-negative transforming growth factor beta type II receptor decreases matrix metalloproteinase 2 in hepatic stellate cells, and upregulates SKI-like oncogene, which antagonizes TGF-beta signaling. | liver, |
| Adenoviral delivery of dominant-negative transforming growth factor beta type II receptor up-regulates transcriptional repressor SKI-like oncogene, decreases matrix metalloproteinase 2 in hepatic stellate cell and prevents liver fibrosis in rats. | liver, |
| SnoN is involved in differentiation in normal skin and benign and nonmetastatic skin tumors, but plays a proto-oncogenic role in undifferentiated squamous cell carcinoma | skin, |
| SKIL role in the tumorigenesis and metastasis of colorectal cancer.SNHG14 promotes the tumorigenesis and metastasis of colorectal cancer through miR-32-5p/ski-oncogene-like SKIL axis.SKIL is a downstream target gene of miR-32-5p. | colorectal, |
| Negative feedback regulation of TGF-beta signaling by the SnoN oncoprotein. | None |
| Requirement for the SnoN oncoprotein in transforming growth factor beta-induced oncogenic transformation of fibroblast cells. | None |
| TAK1 MAPK kinase kinase mediates transforming growth factor-beta signaling by targeting SnoN oncoprotein for degradation. | None |
| SnoN oncoprotein enhances estrogen receptor-alpha transcriptional activity. | None |
| Data indicate that tripartite motif containing 33 protein TIF1gamma promotes sumoylation of SKI-like proto-oncogene protein SnoN1 and regulates epithelial-mesenchymal transition (EMT). | None |
| TIF1gamma protein regulates epithelial-mesenchymal transition by operating as a small ubiquitin-like modifier (SUMO) E3 ligase for the transcriptional regulator SnoN1. | None |
| Cloning and characterization of the murine homolog of the sno proto-oncogene reveals a novel splice variant. | None |
| Proto-oncogene Sno expression, alternative isoforms and immediate early serum response. | None |