| Molecular cloning, genomic structure and interactions of the putative breast tumor suppressor TACC2. | breast, |
| The tumor suppressor gene hSNF5 was lacking in the malignant rhabdoid tumor of the liver. | liver, |
| SMARCB1/INI1 tumor suppressor gene is frequently inactivated in epithelioid sarcomas. | sarcoma, |
| INI1hSNF5 and PARVG do not seem to be the tumor suppressor genes involved in oligodendroglioma development and progression | glioma, |
| SMARCB1 is a tumor suppressor for schwannomas in the context of familial disease. | schwannomas, |
| Recurrent loss, but lack of mutations, of the SMARCB1 tumor suppressor gene in T-cell prolymphocytic leukemia with TCL1A-TCRAD juxtaposition. | leukemia, |
| Loss of the SNF5 tumor suppressor acting through BRG1 is associated with neoplasms. | neoplasm, |
| A germline missense mutation in exon 2 of SMARCB1 predisposes to multiple meningiomas and a four-hit mechanism of tumour suppressor gene inactivation, involving SMARCB1 and NF2, might be operative in familial multiple meningiomas associated meningiomas. | meningiomas, |
| CAPZB is involved in tumor progression in cases of epithelioid sarcoma (EpiS), irrespective of the INI1 expression, and may be a potential therapeutic target. The paradoxical relationship between the tumor suppressor INI1 and the oncoprotein CAPZB in the pathogenesis of EpiS remains to be clarified | sarcoma, |
| Role of the Sin3-histone deacetylase complex in growth regulation by the candidate tumor suppressor p33(ING1). | None |
| SWI/SNF complex interacts with tumor suppressor p53 and is necessary for the activation of p53-mediated transcription | None |
| Data suggest that one mechanism by which INI1/hSNF5 exerts its tumor suppressor function is by mediating the cell cycle arrest due to the direct recruitment of HDAC activity to the cyclin D1 promoter, causing its repression and G(0)-G(1) arrest. | None |
| Highly penetrant, rapid tumorigenesis through conditional inversion of the tumor suppressor gene Snf5. | None |
| INI1 is a tumor suppressor gene gone awry in malignant rhabdoid tumor cells | None |
| tissues heterozygous for the Ini1 tumor suppressor express levels of protein and message roughly equivalent to the levels observed in wild type counterparts. Compensation of Ini1 is mediated by an increase in the rate of transcription from the promoter. | None |
| Loss of the INI1 tumor suppressor does not impair the expression of multiple BRG1-dependent genes or the assembly of SWI/SNF enzymes. | None |
| The tumor suppressor hSNF5/INI1 modulates cell growth and actin cytoskeleton organization. | None |
| tumor suppressor activity of Snf5 depends on its regulation of cell cycle progression | None |
| ING tumor suppressor proteins are critical regulators of chromatin acetylation required for genome expression and perpetuation. | None |
| SMARCB1 may be a tumor suppressor gene for malignant rhabdoid tumor. | None |