| Association between src-kinases and the polyoma virus oncogene middle T-antigen requires PP2A and a specific sequence motif. | None |
| Critical amino acid substitutions in the Src SH3 domain that convert c-Src to be oncogenic. | None |
| Requirement for Ras/Rac1-mediated p38 and c-Jun N-terminal kinase signaling in Stat3 transcriptional activity induced by the Src oncoprotein. | None |
| v-Src suppresses SHPS-1 expression via the Ras-MAP kinase pathway to promote the oncogenic growth of cells. | None |
| The role of polyamines, Na(+) and K(+) in the formation of triple helices between purine oligonucleotides and the promoter region of the human c-src proto-oncogene. | None |
| Full oncogenic activities of v-Src are mediated by multiple signaling pathways. Ras as an essential mediator for cell survival. | None |
| Regulation of both apoptosis and cell survival by the v-Src oncoprotein. | None |
| The adapter type protein CMS/CD2AP binds to the proto-oncogenic protein c-Cbl through a tyrosine phosphorylation-regulated Src homology 3 domain interaction. | None |
| Induction of p21WAF1/CIP1 and cyclin D1 expression by the Src oncoprotein in mouse fibroblasts: role of activated STAT3 signaling. | None |
| A novel group IIA phospholipase A2 interacts with v-Src oncoprotein from RSV-transformed hamster cells. | None |
| The oncoprotein Tax binds the SRC-1-interacting domain of CBP/p300 to mediate transcriptional activation. | None |
| DNA vaccination against v-src oncogene-induced tumours in congenic chickens. | None |
| The molecular perspective: the src oncogene. | None |
| Activated SRC oncogene phosphorylates R-ras and suppresses integrin activity. | None |
| The molecular perspective: the Src oncogene. | None |
| Increased activity of c-Src and Csk in fibroblasts transformed by v-src oncogene. | None |
| Ten introns interrupting the coding sequence of the mouse src protooncogene were sequenced in Balb C mice. Intron 5 was also sequenced in additional mouse strains because the CA and GT repeat array differences are a new polymorphic marker on chromosome 2. | None |
| c-Src activation by the E5 oncoprotein enables transformation independently of PDGF receptor activation. | None |
| Cloning of the proto-oncogene c-src from rat testis. | None |
| proto-oncogene c-src from rat testis was cloned | None |