| QM, a putative tumor suppressor, regulates proto-oncogene c-yes. | None |
| Src promotes destruction of c-Cbl: implications for oncogenic synergy between Src and growth factor receptors. | None |
| src has a role in promoting destruction of c-Cbl and enabling EGFR to evade desensitization as part of an oncogenesis pathway | None |
| Initiation factor eIF2B not p70 S6 kinase is involved in the activation of the PI-3K signalling pathway induced by the v-src oncogene. | None |
| The v-src oncogene may not be responsible for the increased radioresistance of hematopoietic progenitor cells expressing v-src. | None |
| Mutations in the SH3 domain of the src oncogene which decrease association of phosphatidylinositol 3 -kinase activity with pp60v-src and alter cellular morphology. | None |
| Factor-dependent erythroid cell lines derived from mice transplanted with hematopoietic cells expressing the v-src oncogene. | None |
| Autocrine angiotensin system regulation of bovine aortic endothelial cell migration and plasminogen activator involves modulation of proto-oncogene pp60c-src expression. | None |
| Specific association of the proto-oncogene product pp60c-src with an intracellular organelle, the PC12 synaptic vesicle. | None |
| Activation of phosphatidylinositol-3 kinase by nerve growth factor involves indirect coupling of the trk proto-oncogene with src homology 2 domains. | None |
| Expression of developmentally relevant proteins by rodent embryo CNS cells in vivo and in vitro: proto-oncogene pp60c-src and high molecular weight neurofilament protein. | None |
| v-Src rescues actin-based cytoskeletal architecture and cell motility and induces enhanced anchorage independence during oncogenic transformation of focal adhesion kinase-null fibroblasts. | None |
| Gain-of-function screen identifies a role of the Src64 oncogene in Drosophila mushroom body development. | None |
| EGFR, ErbB2 and Ras but not Src suppress RhoB expression while ectopic expression of RhoB antagonizes oncogene-mediated transformation. | None |
| Depudecin: a novel compound inducing the flat phenotype of NIH3T3 cells doubly transformed by ras- and src-oncogene, produced by Alternaria brassicicola. | None |
| Active Rho is localized to podosomes induced by oncogenic Src and is required for their assembly and function. | None |
| Oncogenic mutations reduce the stability of SRC kinase. | None |
| Inhibition of tyrosine kinase and src oncogene functions by stable erbstatin analogues. | None |
| Influence of acidic residues on substrate specificity of oncogene products pp60v-src and p56lck in vitro. | None |
| P120-GAP associated with syndecan-2 to function as an active switch signal for Src upon transformation with oncogenic ras. | None |