| Overexpression of the far upstream element binding protein 1 in hepatocellular carcinoma is required for tumor growth. | liver, |
| FUBP1 acts as a potential oncogene in clear cell renal cell carcinoma (ccRCC) and may be considered as a novel biomarker or an attractive treatment target of ccRCC. | kidney, |
| oncogenic potential of c-Myc is _switched off_ after apoptosis induction as a consequence of the caspase-mediated cleavage of FBP-1. | None |
| These findings are the first report describing the regulation of alternative splicing of MDM2 mediated by the oncogenic factor FUBP1. | None |
| direct connection between the cellular PI3K/AKT/mTOR signaling pathway, frequently activated in human hepatocarcinogenesis, and the enrichment of oncogenic transcription factors of the FBP family | None |
| elevated FUBP1 promotes NB glycolysis and growth by targeting HIF1alpha rather than N-Myc, suggesting that FUBP1 is a novel and powerful oncogene in the development of NB independent of N-Myc and may have potential in the diagnosis and treatment of NB. | None |
| Identification of FUBP1 as a Long Tail Cancer Driver and Widespread Regulator of Tumor Suppressor and Oncogene Alternative Splicing. | None |