| Phosphorylation of deleted in liver cancer 1 (DLC1) and DLC2 by Akt at the conserved residue points to a common regulatory mechanism of the DLC tumor suppressor family. | liver, |
| DLC2 operates as a tumor suppressor gene in breast cancer via the RhoGTPase pathway | breast, |
| Differential regulation of rho GTPases during lung adenocarcinoma migration and invasion reveals a novel role of the tumor suppressor StarD13 in invadopodia regulation | lung, |
| Results suggest that DLC2 exhibits its tumor suppressor functions in vivo as a GAP specific for RhoA, exerting its effects in suppression of cytoskeleton reorganization, cell growth, cell migration, and transformation. | None |
| Expression profile of the tumor suppressor genes DLC-1 and DLC-2 in solid tumors. | None |
| The present study further describes the role of StarD13 as a tumor suppressor as well as a Rho GAP. | None |
| Study describes STARD13 as a tumor suppressor playing a positive role in cancer motility. | None |
| The tumor suppressor DLC2 and Kif1B are central components of a signaling network that guides spindle positioning, cell-cell adhesion and mitotic fidelity. | None |
| Results indicate that Stard13 acts as a metastasis suppressor rather than a tumor suppressor gene, in Neu oncogene induced mammary tumorigenesis. | None |