| Itch E3 ubiquitin ligase regulates large tumor suppressor 1 stability [corrected]. | None |
| LATS1 tumor suppressor is a novel actin-binding protein and negative regulator of actin polymerization. | None |
| Downstream Hippo molecules STE20-like protein kinases 1/2, large tumor suppressor kinases 1/2, and Yes-associated protein (YAP, the effector of the pathway) are phosphorylated, offering novel mechanisms for arrhythmogenic cardiomyopathy pathogenesis. | None |
| Data indicate that tumour suppressor RASSF1A triggers large tumor suppressor kinase 1 (LATS)-CDK2 interaction and restricts CDK2 kinase activity towards BRCA2. | None |
| Findings suggest that polyomavirus middle T-antigen (PyMT) activates the Hippo pathway tumor suppressor Lats in a Src-dependent manner. | None |
| results reported here further support that LATS1/2 act normally as tumor suppressors and loss of their functions contributes to human cancer development | None |
| Study demonstrates that, in 3 different murine syngeneic tumor models (B16, SCC7, and 4T1), loss of the Hippo pathway kinases LATS1/2 (large tumor suppressor 1 and 2) in tumor cells inhibits tumor growth; data indicate a new paradigm for how tumor immunogenicity is regulated through the Hippo signaling pathway in tumor cells. | None |
| DLG5 inhibits the association between MST1/2 and large tumor suppressor homologs 1/2 (LATS1/2), uses its scaffolding function to link MST1/2 with MARK3, and inhibits MST1/2 kinase activity | None |
| Data indicate that large tumor suppressor kinase 1 (LATS1) might participate in reactive astrogliosis after spinal cord injury. | None |
| Monitoring Hippo Signaling Pathway Activity Using a Luciferase-based Large Tumor Suppressor (LATS) Biosensor | None |
| miR-29c-3p regulates the methylation of large tumor suppressor gene 1 (LATS1) by DNMT3B, and abnormal methylation of LATS1 inactivates Hippo signaling pathway. | None |
| MiR-16a Regulates Milk Fat Metabolism by Targeting Large Tumor Suppressor Kinase 1 (LATS1) in Bovine Mammary Epithelial Cells | None |